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Übersetzung für 'β-amyloid' von Englisch nach Deutsch

beta amyloid <β-amyloid> [amyloid beta]

Beta-Amyloid {n} <β-Amyloid>med.

• Beta-Amyloid {n} <β-Amyloid> = beta amyloid <β-amyloid> [amyloid beta]

• These genetic variations change the degradation pathways of misfolded protein contributing misfolded β-amyloid accumulation and plaque formation.
• However, recent studies demonstrate that LILRB2 is a β-Amyloid receptor and may contribute to synaptic loss and cognitive impairment in Alzheimer's disease.
• These genetic variations change the degradation pathways of misfolded protein contributing misfolded β-amyloid accumulation and plaque formation.
• One of the more prominent signs of Alzheimer's disease is the accumulation of β-amyloid proteins in the brain.
• The difference in observations between mice ("CCR2" depletion causes cognitive decline) and humans (higher "CCR2" associated with lower cognitive function) could be due to increased "demand" for macrophage activation during cognitive decline, associated with increased β-amyloid deposition (a core feature of Alzheimer's disease progression).

• Additionally it relieves the inhibition of both α2β2 and α4β2 nicotinic acetylcholine receptors by β-Amyloid (1–42) Peptide.
• In Alzheimer's disease, there is a progressive accumulation of β-amyloid protein (Aβ) in brain.
• MeOTf has been used extensively in the production of Pittsburgh Compound B, which first allowed β-amyloid plaques to be imaged in a living brain.
• These genetic variations alter β-amyloid homeostasis contributing to its accumulation and plaque formation.
• rather than β-secretase, the toxic protein β amyloid is not produced.

• Other ligands include: β-amyloid, heat shock proteins, surface molecules of Gram-positive and Gram-negative bacteria, hepatitis C virus.
• Multiwall nanotubes with a diameter of 80–300 nm, made of dipeptides from the diphenylalanine motif of Alzheimer's β-amyloid peptide are made by this method.
• Additionally, variations in the locational characteristics of "β"-amyloid precursor proteins seen in Alzheimer’s disease are connected to Hirano bodies.
• β-amyloid, a peptide involved in Alzheimer's disease, is also seen in this protein accumulation.
• It is intriguing to note that the increase in ripple amplitude is not seen in adult male TgF344-AD rats which express human β-amyloid precursor protein (with the Swedish mutation) and human presenilin-1 (with a Δ exon 9 mutation).

• β-Amyloid is a peptide of 39 to 43 amino acids. The isoforms with 40 and 42 amino acids (Aβ40/42) are the main constituents of amyloid plaques in the brains of Alzheimer's disease patients.
• Abnormal PLD activity has also been suspected in Alzheimer's disease, where it has been observed to interact with presenilin 1 (PS-1), the principal component of the γ-secretase complex responsible for the enzymatic cleavage of amyloid precursor protein (APP).
• Gliosis and glial scarring occur in areas surrounding the amyloid plaques which are hallmarks of the disease, and postmortem tissues have indicated a correlation between the degree of astrogliosis and cognitive decline.